Philipp Kanske

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Philipp Kanske

Philipp Kanske. Mitglied von bis Fach: Psychologie/​Neurowissenschaften. Kontakt Technische Universität Dresden Institut für Klinische Psychologie. Philipp Kanske (* in Dresden) ist ein deutscher Psychologe und Neurowissenschaftler. Er ist Professor an der Technischen Universität Dresden. Philipp Kanske ist ein deutscher Psychologe und Neurowissenschaftler. Er ist Professor an der Technischen Universität Dresden. Seine Forschungsschwerpunkte sind die neuronalen Grundlagen von Emotion, Emotionsregulation und Emotionsverstehen sowie.

Philipp Kanske

Prof. Dr. Philipp Kanske. Research Associate. Abteilung Soziale Neurowissenschaft · +49 · [email protected] · [email protected]​ Philipp Kanske (* in Dresden) ist ein deutscher Psychologe und Neurowissenschaftler. Er ist Professor an der Technischen Universität Dresden. Das Ziel des Max-Planck-Instituts für Kognitions- und Neurowissenschaften ist die Erforschung von kognitiven Fähigkeiten und Gehirnprozessen beim.

Philipp Kanske Prof. Dr. Philipp Kanske, Director of the Institute Video

Tania SINGER (Max Planck Institute): \

Philipp Kanske is Professor for Clinical Psychology and Behavioral Neuroscience at Technische Universität Dresden and Research Associate at the Max Planck Institute for Human Cognitive and Brain Sciences in Leipzig. He explores the emotional and cognitive processes that enable social behavior and their alterations in psychopathology. Philipp Kanske (* in Dresden) is a German psychologist and neuroscientist. He is professor at Technische Universität Dresden. His research focuses on the neuronal foundations of emotion, emotion regulation and emotion understanding (empathy, perspective-taking), as well as their changes in mental disorders. Philipp Kanske 1, Anne Böckler 2 3, Tania Singer 4 Affiliations 1 Department of Social Neuroscience, Max Planck Institute for Human Cognitive and Brain Sciences, Stephanstr. 1a, , Leipzig, Germany. [email protected] Philipp Kanske: Das soziale Gehirn. Public · Hosted by Deutsches Hygiene-Museum and Die Junge Akademie. Interested. Invite. clock. Thursday, March 12, at Philipp Kanske The emotional matching paradigm, introduced by Hariri and colleagues in , is a widely used neuroimaging experiment that reliably activates the amygdala. 22 rows · Philipp Kanske. Technische Universität Dresden. Bestätigte E-Mail-Adresse bei tu . 3/22/ · > Philipp Kanske. Related. The Brain from Inside Out: Kavli Keynote Address Shines Light on Cognition. New York University’s György Buzsáki proposes that our newborn brains are filled with largely random patterns, which he refers to as an “inside-out” framework. Philipp Kanske; Scientific reports. Published on 08 Oct 0 views XX downloads; XX citations; Perspective Change and Personality State Variability: An Argument for the Role of Self-Awareness and an Outlook on Bidirectionality (Commentary on . Philipp Kanske ist ein deutscher Psychologe und Neurowissenschaftler. Er ist Professor an der Technischen Universität Dresden. Seine Forschungsschwerpunkte sind die neuronalen Grundlagen von Emotion, Emotionsregulation und Emotionsverstehen sowie. Philipp Kanske ist Professor für Klinische Psychologie und Behaviorale Neurowissenschaft an der Technischen Universität Dresden und Research Associate. Philipp Kanske is Professor for Clinical Psychology and Behavioral Neuroscience at Technische Universität Dresden and Research Associate at the Max Planck. Philipp Kanske (* in Dresden) ist ein deutscher Psychologe und Neurowissenschaftler. Er ist Professor an der Technischen Universität Dresden.
Philipp Kanske

Sunny Valentina Pahde an seinen Bruder Chris Waterfront Philipp Kanske und Als Luis Ahrens steigt der Neu-Berliner in die Serie ein. - Prof. Dr. Philipp Kanske, Director of the Institute

Seine Forschungsschwerpunkte sind die neuronalen Grundlagen von Fleabag Staffel 3Emotionsregulation und Emotionsverstehen EmpathiePerspektivenübernahme sowie deren Veränderungen bei psychischen Störungen.

Artikel verbessern Neuen Artikel anlegen Autorenportal Hilfe Letzte Änderungen Kontakt Spenden. Buch erstellen Als PDF herunterladen Druckversion.

English Links bearbeiten. Head of the group Prof. Name Prof. Philipp Kanske Send Email. Contact Information Address work Visitor Address: Chair for Clinical Psychology and Behavioral Neuroscience, Room Chemnitzer Str.

Kanske studied psychology at Technische Universität Dresden and at the University of Oregon with scholarships of Evangelisches Studienwerk Villigst and the Fulbright Commission.

He received his doctorate with summa cum laude from Leipzig University in He then worked as a postdoctoral researcher at the Central Institute of Mental Health in Mannheim and at Heidelberg University , where he did his habilitation in From to he was a group leader at the Max Planck Institute for Human Cognitive and Brain Sciences in Leipzig.

Nevertheless, one of the most important hubs seems to be the amygdala, a group of nuclei in medial temporal cortex LeDoux, The vast neuroimaging literature corroborates this.

This feature selection seems to be highly plastic as shown in cortical blindness with complete loss of visual evoked event-related electroencephalographic responses Hamm et al.

Their input is essential for conscious processing of emotions, which requires involvement of cortical regions such as orbitofrontal and anterior cingulate cortex.

The anterior cingulate cortex is very consistently activated in experimental tasks with emotional stimuli Dalgleish, While the ventral portion is supposed to mainly underlie implicit regulatory processes of emotion generating limbic regions Etkin et al.

The anterior insula, in particular, is a center for interoceptive re-represention and as such discussed as critical for the generation of subjective feeling Craig, It is also crucial for understanding and sharing the emotions of others Singer, Indexing the sustained effects of emotion on stimulus processing, a late positive potential is also consistently observed to be increased for emotional over neutral material Olofsson et al.

It typically starts around ms after stimulus onset and peaks between and ms. Conversely, emotions also influence cognitive processing Pessoa, This Section discusses these reciprocally interactive processes.

Other, more adaptive regulation strategies have been thoroughly investigated as well, the most prominent example being reappraisal.

It is a strategy of cognitive change, referring to the reinterpretation of the meaning of a situation, which yields an altered emotional response Kalisch, A strategy drawing more on attentional control is distraction from the emotional aspects of a scene.

Interestingly, some mechanisms have emotion regulatory effects, even though they do not primarily aim at modulating the emotional experience.

Affect labeling, for example, which refers to using words to characterize feelings or the emotional aspects of an event, reduces the subjective and physiological intensity of emotional responses Lieberman et al.

While these strategies rely on very different psychological mechanisms, their modulating effects on emotional processing are very similar. This also seems to be the case for the neural mechanisms underlying emotion regulation.

Regarding its neural underpinnings, the concept of emotion regulation translates into inhibitory and facilitatory interactions of control and regulated networks that are relatively well understood.

As discussed in the previous section, some brain regions are crucially involved in the generation of emotion; the most prominent example is the amygdala.

This effect is driven by a network of brain regions, critically including dorsolateral and medial prefrontal cortices. Such investigations, however, would be of great clinical importance to identify those strategies that patients can not apply adequately as treatment targets and those techniques that patients are not impaired in as resources.

The neural mechanisms underlying facilitatory influences are partly separate for different functions, because they involve the networks that are specific to the cognitive task at hand.

For memory enhancement, this includes structures in the medial temporal lobe, the hippocampus and associated parahippocampal regions Dolcos et al.

With regard to interfering effects of emotion, they also involve the particular neural networks involved in the specific function that is impaired.

However, the evidence regarding the nature of this influence is inconsistent. Some studies reported an increase in activation of task-relevant brain regions, potentially indicating compensatory activation to preserve goal-directed behavior Blair et al.

A shortcoming of these studies is that they did not directly test if the hyper- and hypo-activations were really located in the neural networks that are essential and specific to the processing of a certain cognitive task.

Therefore, the activation changes due to emotion in these studies may not be directly related to the task, but to another concurrent process.

Sophisticated functional localization of specific networks, before testing the influence of emotion on them would allow better delineation of the mechanisms underlying emotional distractibility.

The clinical relevance lies mainly in the fact that patients with affective disorders are often characterized by cognitive deficits, which might be partially explained by inadequate emotion-cognition interactions.

Typically this has been investigated through interindividual differences in emotional state, for example in the presence of dysphoric mood, or through the induction of an emotional state, for example with the help of affective pictures, music or mental imagery.

Such biased processing has been demonstrated for a range of different cognitive functions. Furthermore, emotion induction increases the interference effects of emotional stimuli on cognitive processing Isaac et al.

The investigations listed above show mood-congruent biasing of valenced stimuli, however, biased information processing may have its most critical impact in the interpretation of ambiguous stimuli.

Rats were given food when they pressed a lever after hearing one specific tone; another tone was paired with aversive white noise, if the rat failed to press a separate lever.

After learning these responses, tones that were intermediate in frequency compared to the other two tones were presented additionally and responses to these ambiguous stimuli were taken as an indicator for a positive bias if the rats pressed the lever associated with food more often or as a negative bias if the lever associated with the white noise was pressed more often.

These animals showed a negative bias in this scenario. Similarly, congenitally helpless rats, that also constitute an animal model of depression, also show a negative bias Enkel et al.

In this study, a pharmacological stressor also had an effect on normal rats and biased rats away from positive responding.

However, in this study the intermediate tones were also reinforced, which renders them non-ambiguous. The study, therefore, did not allow for the detection of an inherent interpretation bias.

Fully adopting this paradigm for research in humans would have the advantage that it enables translational investigation of biased information processing and, thus, testing the neural underpinnings of these mechanisms on multiple levels including the cellular and molecular levels Enkel et al.

The most recent version of the Diagnostic and Statistical Manual of Mental Disorders DSM-5 American Psychiatric Association, lists abnormally and persistently elevated, expansive or irritable mood or depressed mood for most of the day as the A- criteria for manic and depressive episodes, respectively.

These changes are mirrored by generally increased amygdala activity in depression Drevets et al. Such alterations could also be specified for responding to emotional stimuli, for example, using emotional faces that were masked by subsequently presented neutral faces increased activity in the amygdala to all faces, but in particular to fearful ones was found Sheline et al.

Thus, even if the stimuli are not perceived consciously, hyperactivity in emotion generating regions can be observed.

However, there are also studies that did not observe differences in amygdala activity for emotional vs. Also, recent meta- analyses differ in their results regarding amygdala hyperactivation in depression Delvecchio et al.

Also other stimulus categories such as images with emotion-evocative captions elicit elevated amygdala responses in bipolar patients Malhi et al.

As for depression, however, there are also reports of normal amygdala responding to emotional stimuli in bipolar disorder Foland-Ross et al. Meta-analyses seem to yield more support for amygdala hyperactivation in bipolar disorder Delvecchio et al.

A specific of the described studies is that none of them explicitly instructed participants to directly regulate their emotional responses.

This may in part explain the variance in the results, as there are differences in the habitual use of emotion regulation between patients with affective disorders and healthy individuals.

Without explicit regulation instructions, patients and healthy controls may, thus, have dealt differently with arising emotions in the experimental settings.

Interestingly, none of these studies found amygdala hyperactivity to emotional stimuli in the simple viewing condition, but only in the regulation conditions, which is in line with the suggestion that the diverse results regarding amygdala activity may be explained by different implicit application of emotion regulation.

With regard to unipolar depression, the previous studies raised several questions. First, it is unclear if the observed deficit in amygdala regulation through reappraisal generalizes to other regulation strategies or is specific.

Second, all previous studies tested acutely depressed patients. Therefore, it is open, if the deficit is state-dependant or has characteristics of a trait-marker and is still present in remitted patients.

Third, the relation of the observed deficits in amygdala regulation and the self-reports of infrequent use of habitual reappraisal use is unclear.

And fourth, since previous studies tested the regulation of negative emotion only, it is unclear if the deficits are also present for positive emotional stimuli.

At the time the present studies were conducted, there were no published reports on the neural correlates of emotion regulation deficits in bipolar disorder.

Thus the raised questions also apply to bipolar disorder, in addition to the more fundamental question if there are impairments at all in experimental settings.

As it has been consistently shown that emotional distracters have a particularly strong impact on cognitive processing when compared to neutral distracters Dolcos et al.

Several studies investigated cognitive task performance in the presence of emotional distracters. In these studies, emotion did not affect color naming reaction times in patients and in comparison subjects, suggesting that task processing was not directly influenced.

Activation increases without behavioral effects were also found in working memory tasks with sad mood induction Deckersbach et al.

In addition to the lack of behavioral differences between patients and controls, these studies could also not clarify whether the observed hyperactivations reflect altered processing of the cognitive tasks e.

Addressing this issue would require the independent definition of the relevant task network before studying the influence of emotion on this network.

Better understanding of the nature and causes of cognitive deficits in bipolar disorder is of great importance, as the number of deficits in executive function, attention and memory in these patients is correlated with the level of psychosocial functioning Martinez-Aran, Vieta, Colom, et al.

Previous investigations regarding this question, however, yielded partly inconsistent results. These ratings were significantly related to depression scores.

Also, when participants were asked to describe what outcome they had imagined, these outcomes were evaluated as more negative in the group scoring higher in depression by independent raters.

A critical point may be that the scenarios used in this study were preselected from a larger pool of items on the basis of their ability to separate a high from a low depression group.

Thus, it is not clear whether the results would generalize to other situations. Participants were instructed to imagine a situation triggered by their interpretation of the ambiguous stimulus and startle probes were presented in that time-window.

Startle blink reflexes to the ambiguous words were larger in participants high in depression, suggesting that they interpreted the words more negatively.

Here, the latencies to read aloud neutral or negative target words that followed target-valence- congruent or ambiguous prime sentences were measured.

The group high in depression scores did not show naming facilitation for the negative target words, but rather a pattern that results from an attenuated tendency to impose the more negative interpretations.

The variance in the results of these studies may be due to stimulation and design differences. It seems, therefore particularly promising to try assessing the bias with an indirect measure that does not build on previously established associations, as is the case with affective prime sentences or valenced words.

The alternative experimental approach described in Section 2. Beyond the advantage that adopting this paradigm for human research would have in enabling translational investigation of biased information processing, it is also a design that measures the bias indirectly and without making use of stimuli with pre- established positive or negative associations.

In contrast, these associations are experimentally created and fully controlled. Transferring this paradigm to human research is, therefore, an important next step for improving our understanding of biased information processing and its relevance for affective disorders.

They entwine around the central topic of reciprocal interactions of emotion and cognitive control, their neural correlates and, in particular, their potential alterations in unipolar depression and bipolar disorder.

Furthermore, their role as a vulnerability marker for or as a consequence of bipolar disorder is investigated. The three major topics are, first, how cognitive control can influence emotion.

In contrast to implicit or automatic regulation of emotion Phillips, Ladouceur, et al. This question is only little investigated in affective disorders, with more studies being done on depression Beauregard et al.

Second, increased emotional reactivity in bipolar disorder Almeida et al. Cognitive impairments are a widely observed symptom in bipolar disorder Henin et al.

Because previous investigations yielded partly inconsistent results, new paradigms to address this questions, which also allow for translational investigations, should be developed and validated Enkel et al.

Do euthymic bipolar disorder patients and patients with remitted depression show impairments in voluntary emotion regulation through reappraisal and distraction and what are the neural correlates of such impairments?

Hypothesis: Euthymic patients with bipolar disorder and patients with remitted depression show deficits in regulating their emotions through reappraisal as well as distraction.

This should be reflected in a smaller reduction of their ratings of subjective emotional experience compared to the healthy control participants.

Activity in the regulating control network should also be altered and show either increases or decreases compared to healthy control participants.

Inverse functional coupling between emotion generating and regulating regions is expected in the healthy control participants, but not in the patient groups.

Are potential emotion regulation deficits in bipolar disorder a vulnerability marker for the disorder and present in a population at high-risk to develop bipolar disorder, that is, unaffected first-degree relatives of bipolar disorder patients?

Hypothesis: Unaffected first-degree relatives of bipolar disorder patients show similar deficits in emotion regulation as patients with bipolar disorder when compared to matched healthy control participants, which is reflected in subjective ratings, neural activity in emotion generating and regulating regions, as well as connectivity between the latter two.

Do euthymic bipolar disorder patients show increased emotional distractibility when performing a cognitive task? And are such distraction affects accompanied by changes in the neural correlates of processing the task at hand?

Hypothesis: Euthymic patients with bipolar disorder show enlarged impairment in cognitive task processing when emotional distracters are presented simultaneously when compared to neutral distracters and compared to healthy control participants.

This should be reflected in increased response times and error rates in the emotional distraction condition and altered activity in the neural network underlying processing of the task.

Hyperactivation could indicate compensatory effects, while hypoactivation could reflect a breakdown of task-related activity. Is the potentially increased emotional distractibility in bipolar disorder a vulnerability marker or a consequence of the disorder, that is, is it present in high- risk populations as well or can it only be observed in patients with at least one episode of the illness?

Hypothesis: Unaffected first-degree relatives of patients with bipolar disorder and healthy individuals with hypomanic personality show the same increased emotional distractibility as patients with bipolar disorder, when compared to their healthy matched control participants, which is reflected in behavioral performance and neural activation patterns.

Is biased information processing measurable indirectly with a paradigm adapted from animal research and is a negative bias associated with depression related traits?

Healthy individuals show a small positive bias in that they interpret ambiguous stimuli more often as having negative rather than positive consequences.

However, the higher a person scores on depression-related traits, the more negative the bias will be.

Ambiguity and the particular interpretation of a certain stimulus are also reflected in specific alterations of event-related potentials of the electroencephalogram.

Ambiguous stimuli are expected to yield increased amplitudes of the N and of the late positive potential, while the interpretation of a stimulus as negative or positive should also affect the late positive potential amplitude.

Different populations were tested to investigate the neural correlates of emotional processing in depression and bipolar disorder and to specify potential impairments as vulnerability marker or consequence of bipolar disorder.

The populations included healthy individuals to establish the respective paradigms and to serve as gender-, age- and education matched controls for the clinical groups and the groups at risk to develop bipolar disorder.

Currently remitted patients with unipolar depression and euthymic patients with bipolar disorder type I were tested, as well as two populations at high risk to develop bipolar disorder.

These were unaffected first-degree relatives of patients with bipolar disorder type I, who were unrelated, however, to the patients who participated in the studies, and healthy individuals with hypomanic personality.

Thus, it is a method to psychometrically define increased risk for developing bipolar disorder. The two approaches to defining high risk to develop bipolar disorder therefore complement each other.

The outcome measures of the included studies were on the one hand behavioral responses to obtain indicators of performance speed and accuracy as well as subjective reports of experienced emotion and on the other hand functional magnetic resonance imaging fMRI and event-related potentials ERP of the electroencephalogram to allow conclusions about the neural correlates and timing of neural processes, respectively.

It builds on neurovascular coupling, that is, neuronal activity in a certain region leads to increased flow of oxygenated blood into that region.

In comparison to the neural activity that causes it, BOLD is relatively slow and peaks around seconds after the neural activity this is also greatly variable depending, for example, on the specific region.

In the present work, two experimental strategies were used in fMRI. In the reappraisal condition, participants were instructed to re- evaluate a presented emotional image in order to reduce the elicited emotion.

For example, when presented with an image of a crying child one could think that the child will be comforted soon and, in the long run, everything is going to be fine Gross, In the attentional control condition, participants were also presented with emotional images, but solved arithmetic equations simultaneously Van Dillen et al.

Since the two emotion regulation strategies are psychologically very different, their joint investigation allows a better understanding of emotion regulation in affective disorders.

Second, a two-step fMRI experiment was used to investigate the effects of emotion on cognitive processing. In a first step, the neural network involved in a specific cognitive task, a mental arithmetic task, was identified using an established procedure as a functional localizer Rickard et al.

In a second step, activity in this network was tested again with a mental arithmetic task, now with emotional and neutral distracters presented simultaneously.

Thus, the effect of emotion on this network could be directly specified. Previous studies on emotional distraction effects did not include a functional localizer and could, therefore, not specify whether the observed effects were essential and specific to the task at hand, which may be the reason for the partly inconsistent findings of increased activity Blair et al.

In contrast to fMRI, electroencephalography is an already old technique Berger, ; Caton, It registers electrical fields from the scalp that are elicited by ionic currents in the brain Gall et al.

It is, therefore, a direct measure of neural activity and offers superior temporal resolution compared to fMRI. ERPs are voltage fluctuations of the electroencephalogram that are directly related to sensory, motor, affective, or higher cognitive events or processes and result from averaging time- locked epochs of the electroencephalogram, which cancels out noise and shows the activity that is due to such a specific event.

Amplitude and latency differences of the components in the ERP are informative about the differences in the underlying processes and were used in the present work to study biased information processing in ambiguous situations.

The specific paradigm used in the ERP study was an adaptation of a task used in animals Enkel et al. It presents two different tones, one always associated with a reward and the other with punishment.

Participants need to press a button in order to actually obtain the reward or to avoid the punishment. After these associations are learned, additional tones are presented which are intermediate in frequency and, thus, ambiguous with regard to the potential consequence.

Button presses to these ambiguous stimuli are taken as an indicator of a positive bias if the button to obtain reward is pressed more often and of a negative bias if the button to avoid punishment is pressed more often.

In animals it had been shown that normal rats show a slight positive bias, while congenitally helpless rats, which were used as an animal model for depression, show a negative bias Enkel et al.

After enrichment, the negative bias of helpless rats shows a decrease, suggesting that the procedure might serve as a treatment outcome measure in depression as well Richter et al.

Beyond the potential for translational investigations of biased information processing, probing this particular paradigm also has the advantage that it is an indirect measure and does not build on previously established associations like previous investigations with affective prime sentences, naming latencies of valenced words, or sentence completions, which yielded overall incoherent results Berna et al.

To establish this paradigm for use in affective disorder research we tested a group of healthy individuals and related the effects to inter-individual differences in depression related traits, such as rumination.

The first study established a novel paradigm in healthy individuals that allows testing two different types of cognitive regulation of emotion, reappraisal i.

The second and third study applied this paradigm to patients with remitted depression and euthymic bipolar patients and two groups at high risk to develop bipolar disorder.

How to regulate emotion? Neural networks for reappraisal and distraction. Cerebral Cortex, 21, Neural correlates of emotion regulation deficits in remitted depression: The influence of regulation strategy, habitual regulation use, and emotional valence.

NeuroImage, 61, Impaired regulation of emotion: Neural correlates of reappraisal and distraction in bipolar disorder and unaffected relatives.

Translational Psychiatry. Cerebral Cortex June ; doi Email: philipp. The regulation of emotion is vital for adaptive behavior in a social subjective emotional experience, psychophysiological environment.

Different strategies may be adopted to achieve indicators of emotion such as electrodermal activity and heart successful emotion regulation, ranging from attentional control rate Kalisch et al.

However, emotion as measured with electroencephalography Hajcak there is only scarce evidence comparing the different regulation et al.

Specifically, activation of the amygdala is effects on emotional experience. We, therefore, directly compared reduced during reappraisal.

Functional connectivity analyses reappraisal and distraction in a functional magnetic resonance showed that this reduction in amygdala activation during imaging study with emotional pictures.

In the distraction condition reappraisal is negatively related to activity in a neural network participants performed an arithmetic task, while they reinterpreted of control areas Banks et al.

A recent Downloaded from cercor. Both strategies were successful in reducing frontal cortex dlPFC and dmPFC , the orbitofrontal cortex subjective emotional state ratings and lowered activity in the OFC , and the parietal cortex Kalisch as the most bilateral amygdala.

Direct contrasts, however, showed a stronger important nodes of this network. While both strategies relied on common control areas on a concurrent task, thereby reducing emotional responding.

In contrast, the dorsal anterior cingulate and large et al. A recent study by Van Dillen et al. Functional connectivity patterns of the amygdala demonstrated that amygdala downregulation is related to the activation confirmed the roles of these specific activations for the difficulty of the concurrent task.

More difficult tasks also 2 emotion regulation strategies. The Keywords: affect, amygdala, fMRI, mental arithmetic, PPI study provides some indication that activity in these control areas covaries with amygdala activation, but clear evidence for the connectivity of the amygdala during distraction is still lacking.

Introduction To data, the only study that aimed at comparing reappraisal Cognitively influencing emotional experience is highly relevant and distraction combined reappraisal with a working memory for adaptive social behavior and mental and physical health task McRae et al.

They presented emotional pictures, Eftekhari et al. Different strategies can be applied to and participants reinterpreted the images during reappraisal or regulate emotional responses ranging from attentional control kept a 6-letter string in memory during distraction.

The authors to cognitive change Ochsner and Gross While reported activation of the dmPFC, dlPFC, and inferior parietal attentional control enables the individual to focus away from cortex for both tasks.

Reappraisal yielded additional activations an emotional stimulus distraction , cognitive change yields an in the dmPFC and dlPFC, while distraction additionally acti- altered interpretation of an emotional situation reappraisal.

Interestingly, Both strategies have been shown to successfully modulate the amygdala downregulation was stronger during distraction than subjective emotional state and activation in brain areas relevant reappraisal.

However, we know little regulation strategies to elucidate which parts of an emotion as to whether distraction and reappraisal differ in their effects regulation network are common to reappraisal and distraction on emotional experience and in the neural networks un- and which mechanisms are distinct to each strategy.

Also, the derlying the different regulation strategies McRae et al. A number of more Reappraisal is typically examined by instructing participants specific questions remain: First, do the effects described by to alter their emotional response to images or other types McRae et al.

Here, of stimuli by reinterpreting their meaning Ochsner et al. It has been shown to reliably downregulate generated tasks can regulate emotions.

We, therefore, chose to! The Author Published by Oxford University Press. All rights reserved. Attentional orienting towards emotion: P2 and N ERP effects P Kanske, J Plitschka, SA Kotz Neuropsychologia 49 11 , , EmoPics: Subjektive und psychophysiologische Evaluation neuen Bildmaterials für die klinisch-bio-psychologische Forschung M Wessa, P Kanske, P Neumeister, K Bode, J Heissler, S Schönfelder Zeitschrift für Klinische Psychologie und Psychotherapie 39 Suppl.

Time course of emotion-related responding during distraction and reappraisal S Schönfelder, P Kanske, J Heissler, M Wessa Social cognitive and affective neuroscience 9 9 , , Leipzig affective norms for German: A reliability study P Kanske, SA Kotz Behavior research methods 42 4 , , Effortful control, depression, and anxiety correlate with the influence of emotion on executive attentional control P Kanske, SA Kotz Biological psychology 91 1 , , On the influence of emotion on conflict processing P Kanske Frontiers in Integrative Neuroscience 6, 42 , Conflict processing is modulated by positive emotion: ERP data from a flanker task P Kanske, SA Kotz Behavioural Brain Research 2 , ,

Young Investigator Award of Ponzauner Wigg German Society for Psychophysiology and its Application. This allowed the localisation of the core regions in the Dc Heroes for each of the two social skills. Emotion triggers executive attention: anterior cingulate cortex and amygdala responses to emotional words in a conflict task. Research staff at Heidelberg University, Department of Psychiatry, Section of Experimental Psychopathology and Neuroimaging, Das Streben Nach Glück Netflix. PostDoc at the Central Institute of Mental Health, Institute of Cognitive and Clinical Neuroscience, Mannheim. Social Cognitive and Affective Neuroscience, 8, Hypothesis: Unaffected first-degree Hannas Schlafende Hunde of bipolar disorder patients show Leila Lowfire deficits in emotion regulation as patients with bipolar disorder when compared to matched healthy control participants, which is reflected Jennifer Grey Dirty Dancing subjective ratings, Kindle For Kids activity in emotion generating and regulating regions, as well as connectivity between the latter two. Hautzinger M, Keller F, Kühner C. Button presses to these ambiguous stimuli are taken as an indicator of a positive bias if the Thalia Wiesbaden to obtain reward is pressed more often and of a negative bias if the button to avoid punishment is pressed more often. Wessa M, Linke J. Supplemental data ST1 Mean valence and arousal ratings and standard deviations in parentheses for the picture selection. These cookies Süpiegel not store any personal information. Participants need to press a button in order to actually obtain the reward or to avoid the punishment. Liotti, M. To exclude the possibility that the overlay prevented perception and processing of the contents of the images, two analyses were performed. Werner Straub Award of the Department of Psychology at Technische Universität Dresden. Katya Rubia, PhD, Professor of Cognitive Neuroscience Prof. Psychological Bulletin. Neue Zitationen von diesem Autor. Social Cognitive and Affective Neuroscience.


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